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DE:"Autism spectrum disorders"
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DocumentCharacterization of Gprasp2 knockout mice as a new model for autism spectrum disorders2017

Reference code: PT/FB
Entity holding: BIAL Foundation
Location: S. Mamede do Coronado
Title:
BIAL Foundation Archive
Start date: 1994
History:
The BIAL Foundation was created in 1994 by Laboratórios BIAL in conjunction with the Council of Rectors of Portuguese Universities. BIAL’s Foundation mission is to foster the scientific study of Man from both the physical and spiritual perspectives.
Along the years the BIAL Foundation has developed an important relationship with the scientific community, first in Portugal and after worldwide. Today it is an institution of reference which aims to stimulate new researches that may help people, promote more health and contribute to new milestones to gain access to knowledge.
Among its activities the BIAL Foundation manages the BIAL Award, created in 1984, one of the most important awards in the Health field in Europe. The BIAL Award rewards both the basic and the clinical research distinguishing works of major impact in medical research.
The BIAL Foundation also assigns Scientific Research Scholarships for the study of neurophysiological and mental health in people, arousing the interest of researchers in the areas of Psychophysiology and Parapsychology.
To date the BIAL Foundation has supported 461 projects, more than 1000 researchers, with research groups in twenty-seven countries, resulting, until April 2013, in about 600 full papers, out of which 172 published in indexed international journals with an average impact factor of 3.6 and a substantial number of citations (1665).
Since 1996 the BIAL Foundation organizes the Symposia entitled "Behind and Beyond the Brain", a Forum that gathers well renowned neurosciences speakers and the BIAL Foundation Fellows which are spread around the world.
Classified as an institution of public utility, the BIAL Foundation includes among its patrons the Portuguese President, the Portuguese Universities Rectors' Council and the Portuguese Medical Association.
URL: http://www.bial.com/pt/
Accessibility: By permission

Reference code: PT/FB/BL
Entity holding: BIAL Foundation
Title: BIAL Grants
Start date: 1994
History:
In 1994 the BIAL Foundation launched a programme of science research grants with the aim of encouraging the research into Man’s physical and mental processes, namely in fields still largely unexplored but which warrant further scientific analysis, as Psychophysiology and Parapsychology.
Since its launch, applications to the BIAL grants have been increasing. Up to now 461 projects have been supported, involving more than 1000 researchers from 27 countries.
The approved applications have benefited from grants in amounts comprised between €5,000 and €50, 000. The amount to be granted is fixed by the Scientific board according to the needs of each project.
The supported projects have originated, until April 2013, in about 600 full papers, 172 out of which were published in indexed international journals with an average impact factor of 3.6 and a substantial number of citations (1665).
Among the BIAL Foundation fellows is worth highlighting the presence of scientists from prestigious universities from the United States, United Kingdom, Australia, Russia, Germany, Japan, France, Canada, and many others.
The BIAL grants are promoted biannually.

Reference code: PT/FB/BL-2016
Location: BF-GMS
Title:
2016 Grants
Start date: 2017-01

Reference code: PT/FB/BL-2016-266
Location: BF-GMS
Title:
266 - Early life stress and social hierarchies: The role of cortico-striatal circuits
Duration: 2017-01 - 2021-01
Researcher(s):
João Peça, Joana Guedes, Ana Luísa Cardoso, Mohammed Hussien, Lara Franco, Mário Carvalho
Institution(s): Center for Neuroscience and Cell Biology, University of Coimbra (Portugal)
Contents: Contents:
Application form
Correspondence
Research Funding Agreement
Progress report
Final report
Articles
Author: Peça, J.
Secondary author(s):
Guedes, J., Cardoso, A. L., Hussien, M., Franco, L., Carvalho, M.
Number of reproductions:
1
Keywords:
Animal behavior / Social hierarchy / Stress / Optogenetics / Psychophysiology

Reference code: PT/FB/BL-2016-266.02
Location: BF-GMS
Title:
Characterization of Gprasp2 knockout mice as a new model for autism spectrum disorders
Publication year: 2017
Abstract/Results:
ABSTRACT:
Spectrum Disorders (ASD) and Intellectual Disability (ID) are neurodevelopmental disorders that share common aspects in genetic etiology and phenotypic presentation. Nevertheless, despite a significant number of studies the exact mechanisms triggering these neurodevelopmental disorders and the neuronal circuits disrupted are not completely understood. A recent hypothesis has implicated deficits in synaptic plasticity and spine structure on behavioral and cognitive alterations in patients.
G-protein coupled receptors (GPCRs) are known for their role in the regulation of synaptic signaling. Some of the members of this broad family, include the metabotropic glutamate receptor family (mGluR) which are known for their role in synaptic plasticity and in the mediation of long-term depression. Both these mechanisms have been shown to be deregulated in various neuropsychiatric and neurodevelopmental disorders. Additionally, the regulation and trafficking of mGluRs as well as their intracellular partners have been gaining increased attention. In line with this, the G-protein coupled receptor associated sorting protein (Gprasp) family is an interesting target since Gprasp2 has been implicated in psychiatric disorders. Gprasp2 is also a pleiotropic susceptibility candidate gene implicated in autism and is thought to be involved in the endocytic sorting of G-protein coupled receptors.
To understand the role of Gprasp2 in vivo, a Gprasp2 KO model was recently developed. Here we explore the characterization of this animal model at a behavioral and neuronal morphology level. In our behavioral characterization, we found deficits in terms of memory and cognition in Gprasp2 KO mice pointing towards a defect in terms of hippocampal function. Aligned with these results, decreased dendritic complexity and decreased number of mature spines was also found in the CA1 region of the hippocampus. Changes in mRNA levels were also found in the hippocampus that pointed towards a synaptic signaling deficiency. We also found an increase in body weight and changes in behaviors connected to social dominance and aggression in Gprasp2 KO mice, suggesting a role for this gene in hypothalamic circuit function.
Our data suggest that Gprasp2 mutations may contribute to the pathogenesis of neurodevelopmental disorders, affecting hippocampal dendritic and spine morphology and hippocampal and hypothalamic-mediated behaviors. The Gprasp2 KO mice display deficits consistent with ASD and ID-like behaviors.
Accessibility: Document exists in file
Copyright/Reproduction:
By permisison
Language:
eng
Author:
Pereira, M.
Document type:
Master's thesis
Number of reproductions:
1
Reference:
Pereira, M. (2017). Characterization of Gprasp2 knockout mice as a new model for autism spectrum disorders (Unpublished master´s thesis). Faculdade de Ciências e Tecnologia da Universidade de Coimbra, Portugal.
Indexed document: No
Keywords: Autism Spectrum Disorders / Intellectual disability / mGluR / Gprasp2 / Animal model
DocumentMale-specific features are reduced in Mecp2-null mice: Analyses of vasopressinergic innervation, pheromone production and social behaviour2020

Reference code: PT/FB
Entity holding: BIAL Foundation
Location: S. Mamede do Coronado
Title:
BIAL Foundation Archive
Start date: 1994
History:
The BIAL Foundation was created in 1994 by Laboratórios BIAL in conjunction with the Council of Rectors of Portuguese Universities. BIAL’s Foundation mission is to foster the scientific study of Man from both the physical and spiritual perspectives.
Along the years the BIAL Foundation has developed an important relationship with the scientific community, first in Portugal and after worldwide. Today it is an institution of reference which aims to stimulate new researches that may help people, promote more health and contribute to new milestones to gain access to knowledge.
Among its activities the BIAL Foundation manages the BIAL Award, created in 1984, one of the most important awards in the Health field in Europe. The BIAL Award rewards both the basic and the clinical research distinguishing works of major impact in medical research.
The BIAL Foundation also assigns Scientific Research Scholarships for the study of neurophysiological and mental health in people, arousing the interest of researchers in the areas of Psychophysiology and Parapsychology.
To date the BIAL Foundation has supported 461 projects, more than 1000 researchers, with research groups in twenty-seven countries, resulting, until April 2013, in about 600 full papers, out of which 172 published in indexed international journals with an average impact factor of 3.6 and a substantial number of citations (1665).
Since 1996 the BIAL Foundation organizes the Symposia entitled "Behind and Beyond the Brain", a Forum that gathers well renowned neurosciences speakers and the BIAL Foundation Fellows which are spread around the world.
Classified as an institution of public utility, the BIAL Foundation includes among its patrons the Portuguese President, the Portuguese Universities Rectors' Council and the Portuguese Medical Association.
URL: http://www.bial.com/pt/
Accessibility: By permission

Reference code: PT/FB/BL
Entity holding: BIAL Foundation
Title: BIAL Grants
Start date: 1994
History:
In 1994 the BIAL Foundation launched a programme of science research grants with the aim of encouraging the research into Man’s physical and mental processes, namely in fields still largely unexplored but which warrant further scientific analysis, as Psychophysiology and Parapsychology.
Since its launch, applications to the BIAL grants have been increasing. Up to now 461 projects have been supported, involving more than 1000 researchers from 27 countries.
The approved applications have benefited from grants in amounts comprised between €5,000 and €50, 000. The amount to be granted is fixed by the Scientific board according to the needs of each project.
The supported projects have originated, until April 2013, in about 600 full papers, 172 out of which were published in indexed international journals with an average impact factor of 3.6 and a substantial number of citations (1665).
Among the BIAL Foundation fellows is worth highlighting the presence of scientists from prestigious universities from the United States, United Kingdom, Australia, Russia, Germany, Japan, France, Canada, and many others.
The BIAL grants are promoted biannually.

Reference code: PT/FB/BL-2018
Location: BF-GMS
Title:
2018 Grants
Start date: 2019-01

Reference code: PT/FB/BL-2018-085
Location: BF-GMS
Title:
085 - Role of NT3/TrkC in the regulation of fear
Duration: 2019-03 - 2023-07
Researcher(s):
Mónica Santos
Institution(s): Center for Neuroscience and Cell Biology, University of Coimbra (Portugal)
Contents: Contents:
Application form
Correspondence
Research Funding Agreement
Progress report
Final report
Articles
Language: eng
Author:
Santos, M.
Number of reproductions:
1
Keywords:
Neurotrophin 3 / Fear conditioning / TrkC / Fear extinction / Psychophysiology

Reference code: PT/FB/BL-2018-085.02
Location: BF-GMS
Title:
Male-specific features are reduced in Mecp2-null mice: Analyses of vasopressinergic innervation, pheromone production and social behaviour
Publication year: 2020
URL:
https://link.springer.com/article/10.1007/s00429-020-02122-6#citeas
Abstract/Results: ABSTRACT:
Deficits in arginine vasopressin (AVP) and oxytocin (OT), two neuropeptides closely implicated in the modulation of social behaviours, have been reported in some early developmental disorders and autism spectrum disorders. Mutations in the X-linked methyl-CpG-binding protein 2 (MECP2) gene are associated to Rett syndrome and other neuropsychiatric conditions. Thus, we first analysed AVP and OT expression in the brain of Mecp2-mutant mice by immunohistochemistry. Our results revealed no significant differences in these systems in young adult Mecp2-heterozygous females, as compared to WT littermates. By contrast, we found a significant reduction in the sexually dimorphic, testosterone-dependent, vasopressinergic innervation in several nuclei of the social brain network and oxytocinergic innervation in the lateral habenula of Mecp2-null males, as compared to WT littermates. Analysis of urinary production of pheromones shows that Mecp2-null males lack the testosterone-dependent pheromone darcin, strongly suggesting low levels of androgens in these males. In addition, resident-intruder tests revealed lack of aggressive behaviour in Mecp2-null males and decreased chemoinvestigation of the intruder. By contrast, Mecp2-null males exhibited enhanced social approach, as compared to WT animals, in a 3-chamber social interaction test. In summary, Mecp2-null males, which display internal testicles, display a significant reduction of some male-specific features, such as vasopressinergic innervation within the social brain network, male pheromone production and aggressive behaviour. Thus, atypical social behaviours in Mecp2-null males may be caused, at least in part, by the effect of lack of MeCP2 over sexual differentiation.
Accessibility: Document exists in file
Copyright/Reproduction:
By permission
Language:
eng
Author:
Martínez-Rodríguez, E.
Secondary author(s):
Martín-Sánchez, A., Kul, E., Bose, A., Martínez-Martínez, F. J., Stork, O., Martínez-García, F., Lanuza, E., Santos, M., Agustín-Pavón, C.
Document type:
Article
Number of reproductions:
1
Percentiles:
61.02|0.64
Reference:
Martínez-Rodríguez, E., Martín-Sánchez, A., Kul, E., Bose, A., Martínez-Martínez, F. J., Stork, O., ... Agustín-Pavón, C. (2020). Male-specific features are reduced in Mecp2-null mice: Analyses of vasopressinergic innervation, pheromone production and social behaviour. Brain Structure and Function, 225(7), 2219-2238. https://doi.org/10.1007/s00429-020-02122-6
2-year Impact Factor: 3.270|2020
Times cited: 8|2026-02-12
Indexed document: Yes
Quartile: Q1
Keywords: Aggression / Autism spectrum disorders / Methyl-CpG-binding protein 2 / Nonapeptides / Rett syndrome / Social behaviour
DocumentAbnormal pressure pain, touch sensitivity, proprioception, and manual dexterity in children with autism spectrum disorders2016

Reference code: PT/FB
Entity holding: BIAL Foundation
Location: S. Mamede do Coronado
Title:
BIAL Foundation Archive
Start date: 1994
History:
The BIAL Foundation was created in 1994 by Laboratórios BIAL in conjunction with the Council of Rectors of Portuguese Universities. BIAL’s Foundation mission is to foster the scientific study of Man from both the physical and spiritual perspectives.
Along the years the BIAL Foundation has developed an important relationship with the scientific community, first in Portugal and after worldwide. Today it is an institution of reference which aims to stimulate new researches that may help people, promote more health and contribute to new milestones to gain access to knowledge.
Among its activities the BIAL Foundation manages the BIAL Award, created in 1984, one of the most important awards in the Health field in Europe. The BIAL Award rewards both the basic and the clinical research distinguishing works of major impact in medical research.
The BIAL Foundation also assigns Scientific Research Scholarships for the study of neurophysiological and mental health in people, arousing the interest of researchers in the areas of Psychophysiology and Parapsychology.
To date the BIAL Foundation has supported 461 projects, more than 1000 researchers, with research groups in twenty-seven countries, resulting, until April 2013, in about 600 full papers, out of which 172 published in indexed international journals with an average impact factor of 3.6 and a substantial number of citations (1665).
Since 1996 the BIAL Foundation organizes the Symposia entitled "Behind and Beyond the Brain", a Forum that gathers well renowned neurosciences speakers and the BIAL Foundation Fellows which are spread around the world.
Classified as an institution of public utility, the BIAL Foundation includes among its patrons the Portuguese President, the Portuguese Universities Rectors' Council and the Portuguese Medical Association.
URL: http://www.bial.com/pt/
Accessibility: By permission

Reference code: PT/FB/BL
Entity holding: BIAL Foundation
Title: BIAL Grants
Start date: 1994
History:
In 1994 the BIAL Foundation launched a programme of science research grants with the aim of encouraging the research into Man’s physical and mental processes, namely in fields still largely unexplored but which warrant further scientific analysis, as Psychophysiology and Parapsychology.
Since its launch, applications to the BIAL grants have been increasing. Up to now 461 projects have been supported, involving more than 1000 researchers from 27 countries.
The approved applications have benefited from grants in amounts comprised between €5,000 and €50, 000. The amount to be granted is fixed by the Scientific board according to the needs of each project.
The supported projects have originated, until April 2013, in about 600 full papers, 172 out of which were published in indexed international journals with an average impact factor of 3.6 and a substantial number of citations (1665).
Among the BIAL Foundation fellows is worth highlighting the presence of scientists from prestigious universities from the United States, United Kingdom, Australia, Russia, Germany, Japan, France, Canada, and many others.
The BIAL grants are promoted biannually.

Reference code: PT/FB/BL-2014
Location: BF-GMS
Title:
2014 Grants
Start date: 2015-01

Reference code: PT/FB/BL-2014-385
Location: BF-GMS
Title:
385 - Effects of neurofeedback on functional connectivity and EEG power density
Duration: 2015-03 - 2019-06
Researcher(s):
Pedro Jose Montoya Jimenez, Beatriz Rey Solaz, Inmaculada Riquelme, Miguel Angel Munoz Garcia, Niels Birbaumer
Institution(s): University of Balearic Islands - Research Institute on Health Sciences, Palma (Spain)
Contents: Contents:
Application form
Correspondence
Progress reports
Final report
Articles
Language: eng
Author:
Montoya, P.
Secondary author(s):
Solaz, B., Riquelme, I., Garcia, M., Birbaumer, N.
Number of reproductions:
1
Keywords:
Real-time fMRI neurofeedback / Pain / Insula / Anterior cingulate cortex / Psychophysiology

Reference code: PT/FB/BL-2014-385.05
Location: BF-GMS
Title:
Abnormal pressure pain, touch sensitivity, proprioception, and manual dexterity in children with autism spectrum disorders
Publication year: 2016
URL:
https://www.hindawi.com/journals/np/2016/1723401/
Abstract/Results: ABSTRACT:
Children with autism spectrum disorders (ASD) often display an abnormal reactivity to tactile stimuli, altered pain perception, and lower motor skills than healthy children. Nevertheless, these motor and sensory deficits have been mostly assessed by using clinical observation and self-report questionnaires. The present study aims to explore somatosensory and motor function in children with ASD by using standardized and objective testing procedures. Methods. Tactile and pressure pain thresholds in hands and lips, stereognosis, proprioception, and fine motor performance of the upper limbs were assessed in high-functioning children with ASD (n = 27) and compared with typically developing peers (n = 30). Results. Children with ASD showed increased pain sensitivity, increased touch sensitivity in C-tactile afferents innervated areas, and diminished fine motor performance and proprioception compared to healthy children. No group differences were observed for stereognosis. Conclusion. Increased pain sensitivity and increased touch sensitivity in areas classically related to affective touch (C-tactile afferents innervated areas) may explain typical avoiding behaviors associated with hypersensitivity. Both sensory and motor impairments should be assessed and treated in children with ASD.
Accessibility: Document exists in file
Language:
eng
Author:
Riquelme, I.
Secondary author(s):
Hatem, S. M., Montoy, P.
Document type:
Article
Number of reproductions:
1
Percentiles:
95.03|3.08
Reference:
Riquelme, I., Hatem, S. M., & Montoy, P. (2016). Abnormal pressure pain, touch sensitivity, proprioception, and manual dexterity in children with autism spectrum disorders. Neural Plasticity, 1723401. https://doi.org/10.1155/2016/1723401
2-year Impact Factor: 3.054|2016
Times cited: 110|2026-02-08
Indexed document: Yes
Quartile: Q2
Keywords: Motor control / Proprioception / Tactile sensitivuty / Autism spectrum disorders

Abnormal pressure pain, touch sensitivity, proprioception, and manual dexterity in children with autism spectrum disorders

Abnormal pressure pain, touch sensitivity, proprioception, and manual dexterity in children with autism spectrum disorders
DocumentPsychophysiological and behavioral responses to a novel intruder threat task for children on the autism spectrum2017

Reference code: PT/FB
Entity holding: BIAL Foundation
Location: S. Mamede do Coronado
Title:
BIAL Foundation Archive
Start date: 1994
History:
The BIAL Foundation was created in 1994 by Laboratórios BIAL in conjunction with the Council of Rectors of Portuguese Universities. BIAL’s Foundation mission is to foster the scientific study of Man from both the physical and spiritual perspectives.
Along the years the BIAL Foundation has developed an important relationship with the scientific community, first in Portugal and after worldwide. Today it is an institution of reference which aims to stimulate new researches that may help people, promote more health and contribute to new milestones to gain access to knowledge.
Among its activities the BIAL Foundation manages the BIAL Award, created in 1984, one of the most important awards in the Health field in Europe. The BIAL Award rewards both the basic and the clinical research distinguishing works of major impact in medical research.
The BIAL Foundation also assigns Scientific Research Scholarships for the study of neurophysiological and mental health in people, arousing the interest of researchers in the areas of Psychophysiology and Parapsychology.
To date the BIAL Foundation has supported 461 projects, more than 1000 researchers, with research groups in twenty-seven countries, resulting, until April 2013, in about 600 full papers, out of which 172 published in indexed international journals with an average impact factor of 3.6 and a substantial number of citations (1665).
Since 1996 the BIAL Foundation organizes the Symposia entitled "Behind and Beyond the Brain", a Forum that gathers well renowned neurosciences speakers and the BIAL Foundation Fellows which are spread around the world.
Classified as an institution of public utility, the BIAL Foundation includes among its patrons the Portuguese President, the Portuguese Universities Rectors' Council and the Portuguese Medical Association.
URL: http://www.bial.com/pt/
Accessibility: By permission

Reference code: PT/FB/BL
Entity holding: BIAL Foundation
Title: BIAL Grants
Start date: 1994
History:
In 1994 the BIAL Foundation launched a programme of science research grants with the aim of encouraging the research into Man’s physical and mental processes, namely in fields still largely unexplored but which warrant further scientific analysis, as Psychophysiology and Parapsychology.
Since its launch, applications to the BIAL grants have been increasing. Up to now 461 projects have been supported, involving more than 1000 researchers from 27 countries.
The approved applications have benefited from grants in amounts comprised between €5,000 and €50, 000. The amount to be granted is fixed by the Scientific board according to the needs of each project.
The supported projects have originated, until April 2013, in about 600 full papers, 172 out of which were published in indexed international journals with an average impact factor of 3.6 and a substantial number of citations (1665).
Among the BIAL Foundation fellows is worth highlighting the presence of scientists from prestigious universities from the United States, United Kingdom, Australia, Russia, Germany, Japan, France, Canada, and many others.
The BIAL grants are promoted biannually.

Reference code: PT/FB/BL-2014
Location: BF-GMS
Title:
2014 Grants
Start date: 2015-01

Reference code: PT/FB/BL-2014-348
Location: BF-GMS
Title:
348 - Neural basis of mother-child relationship processes: Neural events, theta dynamics, and oxytocin
Duration: 2015-10 - 2021-09
Researcher(s):
Michael J Crowley, Omri Weisman, Richard M. Pasco Fearon, William Moran, Yael Shmueli-Goetz, Lauren Vazquez
Institution(s): Yale University, New Haven, CT (USA)
Abstract/Results: Contents:
Application form
Correspondence
Progress report
Final report
Articles
Language: eng
Author:
Crowley, M.
Secondary author(s):
Weisman, O., Fearon, P., Moran, W., Shmueli-Goetz, Y., Vazquez, L.
Number of reproductions:
1
Keywords:
Mother-child relationship / Attachment / Oscillations / Oxytocin / Psychophysiology

Reference code: PT/FB/BL-2014-348.03
Location: BF-GMS
Title:
Psychophysiological and behavioral responses to a novel intruder threat task for children on the autism spectrum
Publication year: 2017
URL:
https://link.springer.com/article/10.1007/s10803-017-3195-0
Abstract/Results: ABSTRACT:
We measured skin conductance response (SCR) to escalating levels of a direct social threat from a novel, ecologically-relevant experimental paradigm, the Intruder Threat Task. We simultaneously evaluated the contribution of social symptom severity and behavioral movement. Children with AS group showed less psychophysiological reactivity to social threat than controls across all three phases of the experiment. In the AS group, greater social impairment was significantly associated with reduced SCR. However, movement activity predicted SCR while diagnosis did not. Research and treatment need to account for the complex interplay of emotional reactivity and social behavior in AS. Psychophysiology studies of AS should consider the impact of possible confounds such as movement.
Accessibility: Document exists in file
Language:
eng
Author:
South, M.
Secondary author(s):
Taylor, K. M., Newton, T., Christensen, M., Jamison, N. K., Chamberlain, P., Johnston, O., Crowley, M. J., Higley, J. D.
Document type:
Article
Number of reproductions:
1
Percentiles:
21.06|0.19
Reference:
South, M., Taylor, K. M., Newton, T., Christensen, M., Jamison, N. K., Chamberlain, P., Johnston, O., Crowley, M. J., & Higley, J. D. (2017). Psychophysiological and behavioral responses to a novel intruder threat task for children on the autism spectrum. Journal of Autism and Developmental Disorders, 47(12), 3704-3713. https://doi.org/10.1007/s10803-017-3195-0
2-year Impact Factor: 3.476|2017
Times cited: 5|2026-02-08
Indexed document: Yes
Quartile: Q1
Keywords: Autism spectrum disorders / Social threat / Anxiety / Skin conductance response

Psychophysiological and behavioral responses to a novel intruder threat task for children on the autism spectrum

Psychophysiological and behavioral responses to a novel intruder threat task for children on the autism spectrum